Evernote forcing users to change password after hacking attempt

Evernote, the popular cross-platform note taking and sharing app, has issued a statement about some recent "suspicious activity on the Evernote network". All users will have to change their password, and it seems that user names, and other data that includes the encrypted version of passwords has been accessed. In a letter sent out to users, Evernote says the following:

The investigation has shown, however, that the individual(s) responsible were able to gain access to Evernote user information, which includes usernames, email addresses associated with Evernote accounts, and encrypted passwords. Even though this information was accessed, the passwords stored by Evernote are protected by one-way encryption. (In technical terms, they are hashed and salted.)

While our password encryption measures are robust, we are taking steps to ensure your personal data remains secure. This means that in an abundance of caution, we are requiring all users to reset their Evernote account passwords. Please create a new password by signing into your account on evernote.com.

After signing in, you will be prompted to enter your new password. Once you have reset your password on evernote.com, you will need to enter this new password in other Evernote apps that you use. We are also releasing updates to several of our apps to make the password change process easier, so please check for updates over the next several hours.

As we've seen recently, there's a rash of coordinated attempts to hack the big players in online services. Hopefully Evernote's encryption methods are solid, but having users change their password at log in is a great way to keep everyone safe. Visit Evernote's blog for more information.

Source: http://feedproxy.google.com/~r/androidcentral/~3/JIk1vtXOPfQ/story01.htm

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PFT: Broncos use tag on Clady? |? Bears tag Melton

AP

Bills WR Stevie Johnson already is recruiting players like 49ers S Dashon Goldson.? (It?s not tampering unless the team puts the player up to it.)

The Jets have signed three players with Arena League experience.? (Hey, you never know where you?re going to find your next personal punt protector.)

To tag or not to tag, that is the question for the Patriots, as debated by Mike Reiss and Field Yates of ESPNBoston.com.

The Dolphins continue to conduct junior training camps at local elementary schools.? (Sometimes, the fruit just hangs too low.)

The Ravens have a tough decision to make with FB Vonta Leach, who participated in only 42 percent of the snaps in 2012.

Hawaii CB Mike Edwards, a Cleveland native, wants to play for the Browns.

Players who get tagged by the Bengals typically stay only one more year.

Former Steelers RB Carey Davis is pursuing an acting career.

Titans LB Will Witherspoon hopes to play four more years, even though he lost his starting job in 2012.

Colts owner Jim Irsay says that, if the Colts have earned the third pick or lower in 2012, they would have kept QB Peyton Manning.

Jaguars president Mark Lamping talks about the effort to build enhanced video boards at EverBank Field; ?If we don?t keep up, we?re falling behind. We can?t afford to do that in a market this size, because if we do, then we put the franchise at risk,? Lamping said.

Texans LB Brooks Reed could move from the outside to the inside in 2013.? (And it?s time for a question from Gronk:? ?Don?t they already play in a dome??)

Factoring in the trade for QB Alex Smith, the Chiefs will have $7.5 million in cap space, which means they?d have to create more of it to tag WR Dwayne Bowe.

Broncos defensive coordinator Jack Del Rio was scheduled to speak at the MIT Sloan Sports Analytics Conference.? (His comments may or may not have touched upon properly managing water bills.)

Here?s a look at the Chargers who could be cutting their pay in order to help the team clear cap space.

Raiders LS Jon Condo will receive the team?s 2012 Commitment to Excellence Award on March 9.? (The fruit still hangs too low.)

Cowboys CB Brandon Carr says he restructured his contract because he?s a ?team player.?

Giants coach Tom Coughlin reunited with players from his first head-coaching gig, at Rochester Institute of Technology, way back in 1970.

Eagles owner Jeffrey Lurie and his ex-wife are still making movies together.

Suing the league over cap space could mean that the Redskins would never host a Super Bowl.

DE Chris Canty will visit the Packers next week, his agent confirmed.

LB Nick Roach hopes to return to the Bears, at the right price.

LB Justin Durant hopes to return to the Lions, at the right price.

Vikings P Chris Kluwe explains that, when inappropriate questions are posed at the Scouting Combine, the prospective rookie can?t say ?go eff yourself.?

The bounty scandal emerged for the Saints one year ago today.

Former Falcons DE John Abraham still wants to see the team do well, even after it cut him.

Friday?s moves leave the Panthers $3.5 million over the cap, a problem that will be solved as soon as they cut CB Chris Gamble.

With their division rivals dumping a pass rusher, the Buccaneers have even greater incentive to keep DE Michael Bennett off the open market.

ESPN?s Trent Dilfer thinks 49ers (for now) QB Alex Smith will be in even better position in Chiefs coach Andy Reid?s offense.

It?s been 11 years since the Seahawks introduced the uniforms they abandoned a year ago.

Rams COO Kevin Demoff on analytics in football:? ?There is so much in play with respect to the team?s system . . . there is so much covariance in a football game because there are so many other factors.?

Hearing-impaired advocates have sued the Cardinals for failing to provide appropriate accommodations.? (Apparently, the Cardinals aren?t keen on putting ?the crowd is booing . . . again? on the video boards.)

Source: http://profootballtalk.nbcsports.com/2013/03/01/ryan-clady-receives-franchise-tag/related/

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Celebrities That Were Strippers Before Becoming Famous!

Celebrities That Were Strippers Before Becoming Famous!

We previously posted an article telling you about interesting jobs celebrities had before they became famous. Now we are going to take a peek at today’s Hollywood celebrities that started out in their early career as strippers. What stars made it rain? Some might surprise you! Catherine Zeta-Jones Hollywood actress Catherine Zeta-Jones actually worked as ...

Celebrities That Were Strippers Before Becoming Famous! Stupid Celebrities Gossip Stupid Celebrities Gossip News

Source: http://stupidcelebrities.net/2013/02/celebrities-that-were-strippers-before-becoming-famous/

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Pay attention: Youngster demands politicians' attention

Who says you can't fight City Hall? Certainly not 11-year-old David Williams. The young man reprimanded members of the Dallas City Council for not giving him their undivided attention during a meeting.

In a video that went viral, David stands behind a podium, with a room full of strangers to his back. Well, actually, he stands to the side of the podium because he can't see over it. With the microphone as low as it can go, David asks the City Council about its plans for addressing violence in schools.

That's when things get interesting. While David is speaking, he notices that members of the council, instead of paying attention to him, are walking around and chatting. So, he calls them on it. "Do you feel it is acceptable for City Council members to be up and walking around while constituents are addressing them?" he asks.

Boom. That woke 'em up. Dwaine Caraway, a member of the council, praised David for asking such tough questions. "Let me first be apologetic to you," Caraway said. "I do walk a lot around here ... but it is not so respectful to walk around when visitors are speaking, so I will adhere to that as well."

David said that he would like to be on the council when he's an adult. But he's more interested in becoming president (of the United States, not the council). We think he's got a shot.

Source: http://news.yahoo.com/blogs/sideshow/11-old-reprimands-city-council-rudeness-211156121.html

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Woodward on White House: They said 'You'll regret this' (CNN)

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Source: http://news.feedzilla.com/en_us/stories/politics/top-stories/287926892?client_source=feed&format=rss

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Nigeria growth quickens in Q4 2012, oil output falls

ABUJA (Reuters) - Nigeria's economy grew faster in the fourth quarter of last year than the previous quarter but oil output from Africa's biggest producer fell due to theft and flooding, statistics showed on Thursday.

Africa's second largest economy grew 6.99 percent year-on-year in the fourth quarter 2012, up from 6.48 percent in the third quarter, driven by growth in construction, leisure and power sectors, the national bureau of statistics (NBS) said.

GDP grew 6.58 percent for the full year 2012, down from 7.43 percent in 2011, due to a slowdown in developed economies that buy Nigerian oil and to the impact of an Islamist insurgency in the north of the country.

Oil output fell to an average of 2.14 million barrels per day in the fourth quarter 2012, from 2.52 million bpd in the third quarter but strong global prices helped to offset the impact of lower production on the economy.

Production was impacted by outages caused by theft and flooding, the NBS report said. Nigeria is among the world's top 10 crude exporters and holds the ninth biggest gas reserves.

Oil accounts for 80 percent of government revenues and 95 percent of foreign exchange earnings but agriculture is the largest contributor to GDP with a weighting of 40 percent.

Nigeria is growing as an investment destination due to its huge potential consumer population of 160 million and improved fiscal and monetary stability.

Standard and Poor's and Fitch both upgraded Nigeria's credit rating last year and investment in fixed income markets has soared since JP Morgan added the country to its bond index in October. Barclays will add Nigeria to its index next month.

Source: http://news.yahoo.com/nigeria-growth-quickens-q4-2012-oil-output-falls-151608735--business.html

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Study identifies growth factor essential to the most common malignant pediatric brain tumor

Feb. 28, 2013 ? A multi-institutional team led by Massachusetts General Hospital (MGH) researchers has identified a molecular pathway that appears to be essential for the growth and spread of medulloblastoma, the most common malignant brain tumor in children. In their report in the Feb. 28 issue of Cell, they show that blocking this pathway -- which involves interactions between tumor cells and the surrounding tissues -- leads to regression of all four molecular subtypes of medulloblastoma in several mouse models.

"Our finding that a pathway carrying signals from host cells to tumor cells via placental growth factor and its receptor neuropilin 1 is critical to the growth of medulloblastoma, regardless of molecular subtype, strongly supports evaluating antibodies against these proteins as a novel therapeutic approach to this pediatric cancer," says Rakesh K. Jain, PhD, director of the Steele Laboratory for Tumor Biology at MGH and corresponding author of the study.

A highly malignant tumor that originates in the cerebellum, medulloblastoma accounts for about 20 percent of all pediatric brain tumors and is ten times more common in children than in adults. While aggressive treatment with surgery, chemotherapy and radiation significantly improves patient survival, those treatments can have long-term developmental, behavioral, and neurological side effects, particularly in the youngest patients, making the need for less damaging therapies essential.

Impetus for the current investigation began with studies by Peter Carmeliet, MD, PhD, of the Vesalius Research Center in Belgium, a co-author of the current study. Carmeliet found that an antibody against placental growth factor (PlGF) could block angiogenesis in a number of adult tumors. Since PlGF, unlike other angiogenic proteins, is not required for normal postnatal development, Jain and his Steele Lab colleague Lei Xu, MD, PhD, proposed targeting PlGF as anti-angiogenic treatment for pediatric tumors. Matija Snuderl, MD, of the Steele Lab, a co-lead author of the current study, then found that PlGF was highly expressed in all types of medulloblastoma. Other members of Jain's team found that high expression of the P1GF receptor neuropilin 1 (Nrp1) was associated with poor survival in medulloblastoma patients.

To investigate mechanisms behind the potential role of PlGF in medulloblastoma, the MGH investigators collaborated with colleagues in the U.S., Belgium, Canada and Germany. They first confirmed that PlGF is expressed in patient samples of all subtypes of medulloblastoma and that expression of Nrp1 was more significant than that of PlGF's more common receptor, VEGFR1. Experiments in several mouse models revealed that the presence of PlGF is essential for the progression of medulloblastoma and that treatment with several antibodies against the growth factor reduced tumor growth and spread, increasing animal survival even without substantially inhibiting angiogenesis

The researchers were surprised to find that most PlGF was produced by surrounding supportive tissue called stroma and not by the tumor cells. Further investigation revealed that release of the developmental protein Shh (sonic hedgehog) by tumor cells induces expression in nearby stromal cells of PlGF, which then binds to the Nrp1 receptor on tumor cells, leading to further tumor growth. The authors note that therapies that block the interaction between PlGF and Nrp1 are less likely to lead to treatment resistance than are therapies directly targeting mutations that drive tumor growth.

"The importance of tumor-stromal interactions has been recognized for decades, especially the formation of new blood vessels to supply tumors," says Jain, the Cook Professor of Radiation Oncology (Tumor Biology) at Harvard Medical School. "Our discovery of an entirely different way that tumor-stromal interactions drive cancer progression supports the exciting possibility that targeting that pathway in medulloblastoma could be more broadly effective with fewer side effects for patients. Antibodies against both PlGF and Nrp1 have been developed and tested in adult patients. There is hope that they could be safe in pediatric patients, but that needs to be established in clinical trials."

In addition to Snuderl, co-lead authors of the Cell article are Ana Batista, PhD, and Nathaniel D. Kirkpatrick, PhD, of the Steele Lab, and Carmen Ruiz de Almodovar, PhD, of the Vesalius Research Center. Collaborating institutions include Children's Hospital Boston, the Vesalius Research Center, University of Leuven, Belgium; Genentech, Inc.; the University of British Columbia; and University Hospital, M?nster, Germany. Support for this study includes a grant from Hoffmann-La Roche and National Institutes of Health grant R01CA163815. Carmeliet has patent applications for intellectual property related to this study, and Jain is on the boards of trustees of H&Q Healthcare Investors and H&Q Life Science Investors.

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The above story is reprinted from materials provided by Massachusetts General Hospital.

Note: Materials may be edited for content and length. For further information, please contact the source cited above.

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Journal Reference:

1. Matija Snuderl, Ana Batista, Nathaniel?D. Kirkpatrick, Carmen Ruiz?de?Almodovar, Lars Riedemann, Elisa?C. Walsh, Rachel Anolik, Yuhui Huang, John?D. Martin, Walid Kamoun, Ellen Knevels, Thomas Schmidt, Christian?T. Farrar, Benjamin?J. Vakoc, Nishant Mohan, Euiheon Chung, Sylvie Roberge, Teresa Peterson, Carlos Bais, Boryana?H. Zhelyazkova, Stephen Yip, Martin Hasselblatt, Claudia Rossig, Elisabeth Niemeyer, Napoleone Ferrara, Michael Klagsbrun, Dan?G. Duda, Dai Fukumura, Lei Xu, Peter Carmeliet, Rakesh?K. Jain. Targeting Placental Growth Factor/Neuropilin 1 Pathway Inhibits Growth and Spread of Medulloblastoma. Cell, 2013; 152 (5): 1065 DOI: 10.1016/j.cell.2013.01.036

Note: If no author is given, the source is cited instead.

Disclaimer: This article is not intended to provide medical advice, diagnosis or treatment. Views expressed here do not necessarily reflect those of ScienceDaily or its staff.

Source: http://feeds.sciencedaily.com/~r/sciencedaily/top_news/top_health/~3/SQkly6nlu2s/130228124136.htm

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